Rp-camps

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cAMP-elevating agents such as the incretin hormone glucagon-like peptide-1 potentiate glucose-stimulated insulin secretion (GSIS) from pancreatic β-cells. However, a debate has existed since the 1970s.

None of these compounds had an appreciable effect on total cellular cAMP levels, yet the effects of milrinone and amrinone on chloride efflux were blocked by treatment of cells with Rp-cAMPS, a cAMP a.

Results: PGE1 significantly decreased the isometric tension in a concentration-dependent manner, which was partially inhibited by pretreating with a KATP channel inhibitor, glibenclamide (1 μM), or an.

The pharmacological agents described here include nerve growth factor (NGF), pituitary adenylate cyclase-activating polypeptide (PACAP), dexamethasone, forskolin, Rp-cAMPs, and U0126; however, any age.

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The pharmacological agents described here include nerve growth factor (NGF), pituitary adenylate cyclase-activating polypeptide (PACAP), dexamethasone, forskolin, Rp-cAMPs, and U0126; however, any age.

Address all correspondence and requests for reprints to: George G. Holz, PhD, Professor of Medicine and Pharmacology, State University of New York Upstate Medical University, IHP 4310 at 505 Irving Av.

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cAMP-elevating agents such as the incretin hormone glucagon-like peptide-1 potentiate glucose-stimulated insulin secretion (GSIS) from pancreatic β-cells. However, a debate has existed since the 1970s.

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None of these compounds had an appreciable effect on total cellular cAMP levels, yet the effects of milrinone and amrinone on chloride efflux were blocked by treatment of cells with Rp-cAMPS, a cAMP a.

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Results: PGE1 significantly decreased the isometric tension in a concentration-dependent manner, which was partially inhibited by pretreating with a KATP channel inhibitor, glibenclamide (1 μM), or an.

Address all correspondence and requests for reprints to: George G. Holz, PhD, Professor of Medicine and Pharmacology, State University of New York Upstate Medical University, IHP 4310 at 505 Irving Av.